Read below for a better explanation than my knowledge-lacking take.
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Science paper Serotonergic neuron-glioma interactions drive high-grade glioma pathophysiology
- Thread starter TransistorBass
- Start date
Pure science papers to share and discuss.
More bad news for anyone who is unlucky enough to get brain cancer and loves psychedelics.
But on a more serious note for anyone who is wondering, the demonstrated mechanism is tumor promotion, not tumor initiation. So this paper is not implying that the use of psychedelics is in any way related to a increased risk for brain cancer. But if you’re unlucky enough to get brain cancer, psychedelics are a risk factor for accelerating the disease, therefore you should not take them if you’re diagnosed.
So @TransistorBass it has nothing to do with an pathway to brain cancer.
But on a more serious note for anyone who is wondering, the demonstrated mechanism is tumor promotion, not tumor initiation. So this paper is not implying that the use of psychedelics is in any way related to a increased risk for brain cancer. But if you’re unlucky enough to get brain cancer, psychedelics are a risk factor for accelerating the disease, therefore you should not take them if you’re diagnosed.
So @TransistorBass it has nothing to do with an pathway to brain cancer.
Happy to be corrected, posted without sufficient understanding of Scientific papers and their writing.
I was happy to be able to correct you as this would have been a serious problem. Anyway the person who I spoke with explained it to me as in that estrogen is also a driving factor in accelerating breast cancer like androgens are for prostate cancer, but both are also very necessary for healthy living.
I'll edit my initial words with an explanation. 
Well I could be wrong, so let’s see if someone is more informed and willing to take on a different position regarding the paper.I'll edit my initial words with an explanation.
Guessing it can't have helped this poor fella - Terence McKenna - Wikipedia
It figures. There's a clear parallel between the neurogenetic effects of psychedelics and the proliferation of malignant cells. I think it was pretty smart to come up with a study like the one in the paper. Fortunately, most psychedelics aren't endogenous…
As well, I wonder if they'll be looking at the effects that harmala alkaloids might have on the same proliferative model.
Harmalas and DMT feel very mushroom-like on the body-load.
In saying that, are you making an implication that this type of combo might also display a similar effect to that of psilocybin on proliferative glioma and, presumably, other serotonin receptor-driven proliferative diseases?
I'd agree on the body-load observation, including a sudden diuretic effect that both (*)-huasca and psilocybin sometimes display. Which, come to mention it, is another, possibly receptor-related, phenomenon deserving of investigation.
I'd still be interested to see an expansion on your assertion, however. What was the thought process behind it?
Just a feeling based on the body load similarity. I'm an Engineer not a Scientist!
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