What makes the dimethylation of tryptamine so psychoactive? How does this trigger our 5ht2a receptors so effectively when tryptamine doesn't?
I understand that I already have a thread open, which like I hope I'm not cutting forum rules, and I'm not offering much in the way of a post, but I am expecting much in the way of an answer.
I understand that I already have a thread open, which like I hope I'm not cutting forum rules, and I'm not offering much in the way of a post, but I am expecting much in the way of an answer.
