Recently I've found a variety of nuciferine, blue lotus full spectrum and ratio blend extracts.
I have taken to filling capsules with 0.2g of a mix made from 20g of >10% assured full spec, and 20g of unlabelled full spec, and 0.15g max from 20g 80% nuciferine lotus extract
View attachment 111696
Taking 120mg doses of nuciferine alongside psychedelics has been an immense potentiator, smoothing out the experience with a muted euphoric flow/power that feels grounding
Lotus aporphine compounds:
Nuciferine = D4 agonist, D2/D5 parital agonist, DAT inhibitor, antipsychotic, antagonist of psychedelic receptors (trip calmer?), agonism of HT1A and HT6
Apomorphine = D1/D2 agonist, Betablocker, antipsychotic, antiaddictive / parkinsons treatment
Glaucine is another aporphine compound found in yellow horned poppy or lotus
LOTUS APORPHINE COMPOUNDS
YELLOW HORNED POPPY
Contraindications of blue lotus extracts nuciferine / apomorphine include taking alongside prescribed beta blockers - dangerous drop in blood pressure. I have both and know well not to mix but that both are effective in life
The effects to explore of the lotus are welcomingly positive so far with my use of blue lotus extracts, like that of a betablocking stimulant that has a subtle psychedelia which potentiates other substances staggeringly powerfully. The same can be said from all I have heard of glaucine, being a dissociative psychedelic working on traditional HT2A receptors. On its own it has a sedative, waking dreamlike trip. This is strange, as blue lotus is also used as a oneirogen. Another similarity to harmalas, glaucine is an MAO-A inhibitor, making any other seretogenic more potent and longer lasting. Though it is only moderately partial and without MAO-B.
I can only picture blue lotus having a synergistic effect with glaucine, so far it atleast has been immensely effective. My only worry is this is strong stuff already in the lotus family, nuciferine / apomorphine etc. between 80-120mg delivers a powerful effect, and can interact dangerously with things. Comparing a similar dose of glaucine, the moderate MAO-A inhibition is more likely also to have the same severe interactions with many medications or substances, including tyramine hypertensive crisis from cheese.
Potentiation this way when in full can also lead to week long trips, like with harmalas. MAO-A inhibition is what is required for DMT to become orally active.
I can already say blue lotus is effective as a betablocker / psychedelic stimulant/antipsychotic. The calming effect attributed to both for their antipsychotic potential is a major promise for a smooth ride in potentiating low doses of psychedelics. There is a lot of promise here I think in a seemingly unexplored aporphine form of harmala-type compound, with all the risks considering also its opiate related sedative / depressant nature
I have taken to filling capsules with 0.2g of a mix made from 20g of >10% assured full spec, and 20g of unlabelled full spec, and 0.15g max from 20g 80% nuciferine lotus extract
View attachment 111696
Taking 120mg doses of nuciferine alongside psychedelics has been an immense potentiator, smoothing out the experience with a muted euphoric flow/power that feels grounding
Lotus aporphine compounds:
Nuciferine = D4 agonist, D2/D5 parital agonist, DAT inhibitor, antipsychotic, antagonist of psychedelic receptors (trip calmer?), agonism of HT1A and HT6
Apomorphine = D1/D2 agonist, Betablocker, antipsychotic, antiaddictive / parkinsons treatment
Glaucine is another aporphine compound found in yellow horned poppy or lotus
LOTUS APORPHINE COMPOUNDS
YELLOW HORNED POPPY
Contraindications of blue lotus extracts nuciferine / apomorphine include taking alongside prescribed beta blockers - dangerous drop in blood pressure. I have both and know well not to mix but that both are effective in life
The effects to explore of the lotus are welcomingly positive so far with my use of blue lotus extracts, like that of a betablocking stimulant that has a subtle psychedelia which potentiates other substances staggeringly powerfully. The same can be said from all I have heard of glaucine, being a dissociative psychedelic working on traditional HT2A receptors. On its own it has a sedative, waking dreamlike trip. This is strange, as blue lotus is also used as a oneirogen. Another similarity to harmalas, glaucine is an MAO-A inhibitor, making any other seretogenic more potent and longer lasting. Though it is only moderately partial and without MAO-B.
I can only picture blue lotus having a synergistic effect with glaucine, so far it atleast has been immensely effective. My only worry is this is strong stuff already in the lotus family, nuciferine / apomorphine etc. between 80-120mg delivers a powerful effect, and can interact dangerously with things. Comparing a similar dose of glaucine, the moderate MAO-A inhibition is more likely also to have the same severe interactions with many medications or substances, including tyramine hypertensive crisis from cheese.
Potentiation this way when in full can also lead to week long trips, like with harmalas. MAO-A inhibition is what is required for DMT to become orally active.
I can already say blue lotus is effective as a betablocker / psychedelic stimulant/antipsychotic. The calming effect attributed to both for their antipsychotic potential is a major promise for a smooth ride in potentiating low doses of psychedelics. There is a lot of promise here I think in a seemingly unexplored aporphine form of harmala-type compound, with all the risks considering also its opiate related sedative / depressant nature
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When I was coming down, I felt/heard low frequency vibrations coming out of the Earth that I had never encountered before.... Yeah – you're in for quite the trip!
